Page 2 - What is IBAL

What is Ion Biotechnology Aqueous Ligands

IBAL vs. Normal Ionophores

Beyond the Ionophoric ac�on of IBAL driving nutrient uptake to make healthy cells healthier and kill abnormal or anaerobic
cells, many diseases follow an anaerobic fermenta�on process which oxygen will impair thereby sugges�ng a secondary
mechanism for destroying the disease.

Minerals that are not in a bioavailable form will not be able to eliminate or otherwise disable the disease cells because the
minerals cannot pass through the membrane coa�ng the outer surface of the disease cells and/or cannot travel in an
extracellular fashion. As an example, even ionic Zn++ cannot pass through the hydrophobic, lipid bilayer cellular wall on its own.
Under normal circumstances, a natural ionophore, such as querce�n (a flavonoid in) surrounds and masks the ionic zinc’s
charge. With the charge masked the zinc can then pass through the cellular membrane. But there are two reasons that IBAL is
far more efficient than normal ionophores. First, the ca�ons are already contained within the IBAL structure and are ready to
pass into cells. Conversely, dietary zinc must first encounter an ionophore to be usable by cells. Second, even when a substance
can pass through the cellular wall the lipids that make up the wall must be pushed aside. The extremely small structure of IBAL
encounters far less resistance passing through the cellular wall than zinc encapsulated by a much larger ionophore such as
querce�n.
Furthermore, IBAL has been tested to have an oxida�on reduc�on poten�al (ORP) of 450+mV and an oxygen radical
absorbance capacity of 1026 μmol TE. In comparison, Vitamin C is perhaps the most famous an�-oxidant and has only 133 μ
mol TE. These extremely high values further illustrate IBAL’s an�-inflamma�on proper�es.
One of the actual mechanisms IBAL uses to reduce inflamma�on is its ability to act as a Superoxide Dismutase (SOD) mimic to
oxidize Radial Oxygen (AKA, Free Radicals or Oxida�ve Stress) into O2. This elimina�on of free radicals helps reduce
inflamma�on and the damage being caused, returning the body to homeostasis.

One of the specific free radical elimina�on mechanisms involves Cu++ dona�ng an electron being reduced to Cu+. The resultant
Cu+ is also the cri�cal component required to complete the Electron Transport Chain, the third and final cellular metabolism
stage, which generates 90% of the ATP (cellular energy) from the aerobic processes. Glycolysis produces a net 2 ATP; the Krebs
Cycle produces another 2 ATP, and the Electron Transport Chain produces the remaining 34 ATP. Sufficient Cu+ is required for
aerobic cells to maximize their energy produc�on which IBAL may help supply.

Further Modes of Ac�on are s�ll under inves�ga�on such as the polarity of the IBAL cons�tuents a�er it has disassociated
driving it toward oxida�ve stress and inflamma�on as well as the presence of many other elements necessary to life such as
Sulfur, Amines, and Molecular Hydrogen. Some of the reac�ve species of oxygen, nitrogen, and sulfur are also heavily involved
in cell signaling normaliza�on. There is evidence of significant heavy metal chela�on effects from long-term IBAL product use.

IBAL is very efficient!

If I’m lucky I might find
IBAL IONOPHORE minerals so I can deliver NORMAL IONOPHORE
them inside a cell.
I already have
minerals with me
so I’m ready to go!
Zn++
Zn++
OUTSIDE Zn++

I’m also very small
CELLULAR Zn++ so it’s easy to get I’m big so it’s
MEMBRANE hard to make it
through here. through here.

INSIDE
That was easy!
Zn++
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